Deep Vein Thrombosis

Immune Factors in Deep Vein Thrombosis Initiation

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Conceptual model. We hypothesized that psoriasis, psoriatic arthritis (PsA) and rheumatoid arthritis (RA) are associated with venous thromboembolism (VTE) as a result of local and/or systemic inflammation which then triggers prothrombotic pathways. Additional risk factors for VTE are also shown and were adjusted for in our analyses; they may be directly related to the risk for VTE or may act as confounders. In this model, we hypothesize local and/or systemic inflammation is a mediator or on the causal pathway between psoriasis or inflammatory arthritis and VTE. This hypothesis was not tested in this study. NSAID, non-steroidal anti-inflammatory drugs; COPD, chronic obstructive pulmonary disease; CKD, chronic kidney disease.
Aims To determine the risk of venous thromboembolism (VTE) defined as the combined endpoint of deep venous thrombosis (DVT) and pulmonary…
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Inflammation is a risk factor for clotting. RA patients appear to be at higher risk than psoriatic patients.

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